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February 1, 1991Journal of VirologyOpen Access

Herpes simplex virus transactivator ICP4 operationally substitutes for the cellular transcription factor Sp1 for efficient expression of the viral thymidine kinase gene

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Authors

AIAnthony N. ImbalzanoUniversity of Massachusetts Chan Medical SchoolDCDonald M. CoenHarvard UniversityNDNeal A. DeLucaUniversity of Pittsburgh

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Imbalzano et al. (1991) studied this question.

synapsesocial.com/papers/6a844154c2c8a33f31f7a29chttps://doi.org/10.1128/jvi.65.2.565-574.1991
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Separation of primary structural components conferring autoregulation, transactivation, and DNA-binding properties to the herpes simplex virus transcriptional regulatory protein ICP41989 · 111 citations
  2. 2Physical and functional domains of the herpes simplex virus transcriptional regulatory protein ICP41988 · 234 citations
  3. 3Delineation of regulatory domains of early (beta) and late (gamma 2) genes by construction of chimeric genes expressed in herpes simplex virus 1 genomes.1989 · 44 citations
  4. 4ICP4-binding sites in the promoter and coding regions of the herpes simplex virus gD gene contribute to activation of in vitro transcription by ICP41989 · 49 citations
  5. 5Isolation and characterization of deletion mutants of herpes simplex virus type 1 in the gene encoding immediate-early regulatory protein ICP41985 · 710 citations