152 was responsible for the abnormal movements, as similar CT findings have been reported in other cases of hemidystonia.26 In the necropsied cases partial destruction of the caudate nucleus and the putamen has been described.1 2 Why there is a delay between the occur- rence of the hemiplegia and the onset of the abnormal movements, which frequently appear after the weakness has improved, is unknown. It has been proposed that aber- rant central nervous system sprouting sub- sequent to a non progressive cerebral insult is responsible for this delay.3 In the present case as well as in previous ones, the chrono- logical progression with the onset of dystonic symptoms after the improvement of the hemiplegia, and subsequent worsening of the movements, would be in keeping with this hypothesis.
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Cooper et al. (1988) studied this question.
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