The site of action of the postemergence graminicide, diclofop‐methyl (DM), in susceptible plants is possibly the plasmalemma. Indole‐acetic acid (IAA)‐ and fusicoccin (FC)‐induced net proton excretion in Avena coleoptiles was inhibited by the free acid, diclofop. However, net proton excretion recovered within 2 h when 2,4‐dichlorophenoxy acid (2,4‐D) was added simultaneously with diclofop. Diclofop depolarized the membrane potential (Em) within 12 min but the Em recovered within 30 min when diclofop was removed and replaced with either IAA or 2,4‐D. The inhibition of IAA‐induced coleoptile growth by DM and the membrane effects of its acid, diclofop, were partially reversed by 2,4‐D if it was added shortly after treatment of the tissue. These results are consistent with the reversal of DM injury in whole plants with 2,4‐D.
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Shimabukuro et al. (1989) studied this question.
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