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July 1, 1992Proceedings of the National Academy of SciencesOpen Access

A dominant mutation in the Wilms tumor gene WT1 cooperates with the viral oncogene E1A in transformation of primary kidney cells.

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Authors

Daniel A. Haber
Daniel A. HaberThe Wistar InstituteHTH. T. Marc TimmersUniversity of FreiburgJPJerry PelletierMcGill University

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Haber et al. (1992) studied this question.

synapsesocial.com/papers/6a85a2054f2eb4dbc91e6673https://doi.org/10.1073/pnas.89.13.6010
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Alternative splicing and genomic structure of the Wilms tumor gene WT1.1991 · 628 citations
  2. 2Analysis of the rat JE gene promoter identifies an AP-1 binding site essential for basal expression but not for TPA induction1990 · 82 citations
  3. 3Isolation, characterization, and expression of the murine Wilms' tumor gene (WT1) during kidney development.1991 · 247 citations
  4. 4Suppression of Tumorigenicity in Wilms Tumor by the p15.5-p14 Region of Chromosome 111991 · 123 citations
  5. 5Binding of the Wilms' Tumor Locus Zinc Finger Protein to the EGR-1 Consensus Sequence1990 · 583 citations