Histrionicotoxin alters the voltage and time dependence of the endplate current in frog muscle by increasing the affinity between the toxin and the ACh receptor-ionic channel complex.
No takes yet. Share an insight, caveat, or question.
Histrionicotoxin modulates frog endplate currents; leaves open its utility for probing ACh receptor channels in mammalian models.
Masukawa et al. (1978) studied this question.
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: