Cells of an Escherichia coli mutant defective in heme biosynthesis (hemA) grown under conditions for respiration deficiency (grown in the absence of aminolevulinic acid) have 20% of the number of specific colicin Ia receptors found in cells of the same strain grown under conditions for respiration competency (grown in the presence of aminolevulinic acid).
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Gilchrist et al. (1976) studied this question.
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