Chlamydia trachomatis; infection; heat shock proteins hronic chlamydial infections have been documented in numerous works 1,z as silently evolving diseases. Evidence of resistance to antibiotic therapy, as demonstrated by positive cultures as well long after treatment in infertile women, 3,4 as positive chlamydial antigens or nucleic acids in culture-negative pelvic samples of infertile womens and the discovery of chlamydial RNA and inflammatory cytokines transcripts in tubes 6 or joints, 7 have led investigators to consider persistence of the infectious material responsible for the acquisition of chronicity. So far, animal models in numerous works have proved the persistence of
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Askienazy-Elbhar et al. (1999) studied this question.
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