T HE theory that ingestion of hot substances may act as a primary etiological or secondary irritative factor in diseases of the stomach has now been extant for many years, without sufficient co-ordinated investigation to prove or disprove its validity. The purposes of the present article are (I ) to document the suggested mechanisms by which local heat may play a part in gastric disease, (2) to present further studies, and
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Davis et al. (1949) studied this question.
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