Abstract —Rabbit vagus nerves and nodose ganglia were incubated in vitro for up to 24 h in two‐compartment chambers. After the introduction of [ 3 H]leucine or [ 3 H]fucose to the ganglion compartments a rapid anterograde axonal transport of labelled proteins or glycoproteins occurred at rates of 330 ± 44 mm/day and 336 ± 30 mm/day respectively. Accumulation of [ 3 H]leucine‐labelled proteins proximal to a ligature on the nerve was unaffected by a delay of up to 6 h between removal of the nerve and labelling in vitro. Accumulation was prevented by inhibition of protein synthesis in the ganglion but not in the axon and was inhibited in a graded manner by colchicine.
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McLean et al. (1975) studied this question.
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