Answers are grounded in this paper and cite their sources.
did not undergo normal PIN rearrangements, suggesting that the proteins encoded by these autism spectrum disorder-linked genes serve as structural hubs for synaptic homeostasis. Our approach demonstrates how changes in the protein content of synapses during homeostatic plasticity translate into functional PIN alterations that mediate changes in neuron excitability.
No takes yet. Share an insight, caveat, or question.
Heavner et al. (2021) studied this question.
Synapse has enriched 10 related papers. Consider them for comparative context: