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April 29, 2011PLoS ONEOpen Access

Expression of Mutant or Cytosolic PrP in Transgenic Mice and Cells Is Not Associated with Endoplasmic Reticulum Stress or Proteasome Dysfunction

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Authors

EQElena QuaglioMario Negri Institute for Pharmacological ResearchERElena RestelliHuman TechnopoleAGAnna GarofoliMario Negri Institute for Pharmacological Research

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Quaglio et al. (2011) studied this question.

synapsesocial.com/papers/6a886b93aadaab60564fd1fbhttps://doi.org/10.1371/journal.pone.0019339
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Cytosolic Prion Protein (PrP) Is Not Toxic in N2a Cells and Primary Neurons Expressing Pathogenic PrP Mutations2005 · 121 citations
  2. 2Prion Pathogenesis is Independent of Caspase-122007 · 44 citations
  3. 3Identification of an Intracellular Site of Prion Conversion2009 · 177 citations
  4. 4Transcriptional Analysis Implicates Endoplasmic Reticulum Stress in Bovine Spongiform Encephalopathy2010 · 26 citations