Nicotine caused a fall in primary spermatocyte number together with a retardation of spermatocyte to spermatid conversions process. These effects of nicotine were possibly secondary to increased activity of adrenomedullary catecholamines, since the antispermatogenic effect of nicotine was prevented by phentolamine, a known blocker of catecholamine action at the alpha-receptor level.
No takes yet. Share an insight, caveat, or question.
Patra et al. (2009) studied this question.
Synapse has enriched 5 related papers. Consider them for comparative context: