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November 11, 2011Apmis

Knockdown of c‐Met enhances sensitivity to bortezomib in human multiple myeloma U266 cells via inhibiting Akt/mTOR activity

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Authors

WQWenzhong QueFujian Medical UniversityJCJunmin ChenMacau University of Science and TechnologyMCMa Chuang

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Que et al. (2011) studied this question.

synapsesocial.com/papers/6a89cf5b0e4958e0bcf7d26ehttps://doi.org/10.1111/j.1600-0463.2011.02836.x
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1PI3K/Akt/mTOR pathway as a target for cancer therapy2005 · 472 citations
  2. 2Cyclin D1 Expression Mediated by Phosphatidylinositol 3-Kinase through mTOR-p70S6K-Independent Signaling in Growth Factor-Stimulated NIH 3T3 Fibroblasts1999 · 182 citations
  3. 3Bortezomib for multiple myeloma2006 · 15 citations
  4. 4Apoptosis and autophagy: Regulation of caspase‐9 by phosphorylation2009 · 229 citations
  5. 5Hepatocyte growth factor and its receptor c-met in multiple myeloma1996 · 214 citations