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December 1, 1989DiabetesOpen Access

Prevention of Diabetes in NOD Mice by Injection of Autoreactive T-Lymphocytes

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Authors

EREva‐Pia ReichMerck & Co., Inc., Rahway, NJ, USA (United States)DSDenise ScaringeJYJunji YagiMemorial

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Cite This Study

Reich et al. (1989) studied this question.

synapsesocial.com/papers/6a8a4ed0abe412682d9321e3https://doi.org/10.2337/diab.38.12.1647
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Evidence implicating L3T4 in class II MHC antigen reactivity; monoclonal antibody GK1.5 (anti-L3T4a) blocks class II MHC antigen-specific proliferation, release of lymphokines, and binding by cloned murine helper T lymphocyte lines.1983 · 371 citations
  2. 2Two types of murine helper T cell clone. I. Definition according to profiles of lymphokine activities and secreted proteins.1986 · 7,554 citations
  3. 3T cell-mediated inhibition of the transfer of autoimmune diabetes in NOD mice.1989 · 257 citations
  4. 4Evidence that the Thy‐1 molecule is the target for T cell mitogenic antibody against brain‐associated antigens1983 · 44 citations
  5. 5Cell-interaction molecules on immunocompetent lymphocytes. Development of anti-parent cell-interaction-molecule-receptor reactions in F1 hybrid mice and evidence for a unique F1 hybrid subset of interacting cells.1981 · 4 citations