Population
17 lungs from autopsied patients aged 2 to 60 months with acute or chronic human adenovirus pneumonia
Design
Case_series
Authors
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This histopathological study delineates the distinct cellular and extracellular matrix changes in acute versus chronic human adenovirus pneumonia, highlighting alveolar collapse as a potential mechanism for chronic fibrosis.
This histopathological study delineates the distinct cellular and extracellular matrix changes in acute versus chronic human adenovirus pneumonia, highlighting alveolar collapse as a potential mechanism for chronic fibrosis.
Rosman et al. (1996) studied this question.