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July 15, 2000Journal of Clinical InvestigationOpen Access

Loss of function and inhibitory effects of human CSX/NKX2.5 homeoprotein mutations associated with congenital heart disease

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HKHideko KasaharaPediatric / Congenital CardiologyBLBora LeeInstitut Pasteur KoreaJSJean‐Jacques SchottElectrophysiology

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Kasahara et al. (2000) studied this question.

synapsesocial.com/papers/6a8a700a9be9d02581bdaecbhttps://doi.org/10.1172/jci9860
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Haploinsufficiency ofMSX1: a Mechanism for Selective Tooth Agenesis1998 · 85 citations
  2. 2Mutations in the cardiac transcription factor NKX2.5 affect diverse cardiac developmental pathways1999 · 695 citations
  3. 3Cardiac and Extracardiac Expression of Csx/Nkx2.5 Homeodomain Protein1998 · 158 citations
  4. 4Premature suture closure and ectopic cranial bone in mice expressing Msx2 transgenes in the developing skull.1995 · 225 citations
  5. 5Homeodomain factor Nkx2-5 controls left/right asymmetric expression of bHLH gene eHand during murine heart development.1997 · 327 citations