Key result
This review summarizes mathematical models of muscular dystrophy that characterize molecular, cellular, immunological, and mechanical components implicated in disease progression.
This review provides a comprehensive overview of mathematical and systems biology models for understanding the complex genetic, mitochondrial, and immunological mechanisms driving muscular dystrophy progression.
Models of muscular dystrophy may guide mechanistic research; leaves open clinical translation and validation.
Muscular dystrophy (MD) describes generalized progressive muscular weakness due to the wasting of muscle fibers. The progression of the disease is affected by known immunological and mechanical factors, and possibly other unknown mechanisms. These dynamics have begun to be elucidated in the last two decades. This article reviews mathematical models of MD that characterize molecular and cellular components implicated in MD progression. A biological background for these processes is also presented. Molecular effectors that contribute to MD include mitochondrial bioenergetics and genetic factors; both drive cellular metabolism, communication and signaling. These molecular events leave cells vulnerable to mechanical stress which can activate an immunological cascade that weakens cells and surrounding tissues. This review article lays the foundation for a systems biology approach to study MD progression.
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Cameron et al. (2016) conducted a review in Muscular Dystrophy. This review summarizes mathematical models of muscular dystrophy that characterize molecular, cellular, immunological, and mechanical components implicated in disease progression.
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