A comparison of four host strains for assay of dry T1 phage plaque survival indicates that about 25% of fast electron direct action damage expressed in uvrB/sup -/ hosts is repaired in uvrB/sup +/ hosts. The repair is not associated with a lex or exr gene. In relative amounts, the uvr-repairable damage corresponds with that reported for the T1 phage irradiated by x rays in heavily protected anoxic suspension and assayed for plaque survival on uvr/sup +/ and uvr/sup -/ host strains.
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Donald J. Fluke (1977) studied this question.
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