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February 20, 2017Oncology ReportsOpen Access

Vinblastine differs from Taxol as it inhibits the malignant phenotypes of NSCLC cells by increasing the phosphorylation of Op18/stathmin

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Authors

FSFang ShenDLDan LongTYTing Yu

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Shen et al. (2017) studied this question.

synapsesocial.com/papers/6a8bdaae95f0361a9ebee802https://doi.org/10.3892/or.2017.5469
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Stathmin 1: a protein with many tasks. New biomarker and potential target in cancer2012 · 49 citations
  2. 2Sensitizing B- and T- cell Lymphoma Cells to Paclitaxel/Abraxane–Induced Death by AS101 via Inhibition of the VLA-4–IL10–Survivin Axis2014 · 19 citations
  3. 3Tumor Necrosis Factor-induced Microtubule Stabilization Mediated by Hyperphosphorylated Oncoprotein 18 Promotes Cell Death2000 · 33 citations
  4. 4Stathmin Protein Level, a Potential Predictive Marker for Taxane Treatment Response in Endometrial Cancer2014 · 38 citations
  5. 5Silencing Op18/stathmin by RNA Interference Promotes the Sensitivity of Nasopharyngeal Carcinoma Cells to Taxol and High‐Grade Differentiation of Xenografted Tumours in Nude Mice2016 · 51 citations