Key result
In a 20-year-old woman with left ventricular non-compaction cardiomyopathy, cardiovascular complaints were ultimately attributed to an infectious exacerbation of bronchial asthma and musculoskeletal disorders.
Case Report (n=1)
In patients with rare cardiomyopathies like LVNC, new cardiovascular symptoms should be carefully evaluated to rule out non-cardiac causes such as asthma exacerbation or musculoskeletal issues.
New symptoms in LVNC may reflect non-cardiac causes; leaves open rigorous differential diagnosis in rare cardiomyopathies.
20-year-old woman with diagnosed in childhood left ventricular non-compaction (LVNC) cardiomyopathy with coexisting arthrogryposis was admitted to the Department of Cardiology due to atypical chest pain, worsening of exercise tolerance, bendopnea and symptoms of bronchial asthma exacerbation. The electrocardiogram (ECG) showed changes indicative of ischemia, without their dynamics. Inflammatory parameters, BNP (B-type natriuretic peptide), and myocardial necrosis markers were not elevated. Systolic and diastolic function on echocardiography (ECHO) was assessed as normal. Despite the initially suggestive clinical picture, the correct direction of the differential diagnosis, and consequently the diagnosis, was established after completing the history of comorbidities.
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Rezaei et al. (2022) conducted a case report in Left ventricular non-compaction cardiomyopathy (n=1). Left ventricular non-compaction cardiomyopathy was evaluated. In a 20-year-old woman with left ventricular non-compaction cardiomyopathy, cardiovascular complaints were ultimately attributed to an infectious exacerbation of bronchial asthma and musculoskeletal disorders.
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