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The neurobiological features of Post-Traumatic Stress Disorder (PTSD), a debilitating psychiatric condition triggered by exposure to traumatic events, are associated with enigmatic pathological mechanisms which are becoming clearer as the library of associated literature increases. This article considers the clinical features of PTSD, the neural circuitry at work, and some of the genetic and epigenetic factors contributing to PTSD risk. In addition, this review presents various models proposed to explain the progression of PTSD, including the Synaptic Model of Chronic Stress Pathology, the Network-based Model, and the Dual Pathology Model.
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Jason et al. (2023) studied this question.
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