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May 24, 2019Frontiers in Cellular NeuroscienceOpen Access

Dysfunction of 67-kDa Laminin Receptor Disrupts BBB Integrity via Impaired Dystrophin/AQP4 Complex and p38 MAPK/VEGF Activation Following Status Epilepticus

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Authors

HPHana ParkSCSeo-Hyeon ChoiMKMin-Jeong Kong

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Park et al. (2019) studied this question.

synapsesocial.com/papers/6a8d26bc0ee74fcafa4ac1fahttps://doi.org/10.3389/fncel.2019.00236
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Transcriptome analysis suggests a role for the differential expression of cerebral aquaporins and the <scp>MAPK</scp> signalling pathway in human temporal lobe epilepsy2017 · 71 citations
  2. 2Severe alterations of endothelial and glial cells in the blood‐brain barrier of dystrophic mdx mice2003 · 164 citations
  3. 3Molecular scaffolds underpinning macroglial polarization: An analysis of retinal Müller cells and brain astrocytes in mouse2012 · 39 citations
  4. 4Role of the p38 Mitogen-Activated Protein Kinase/Cytosolic Phospholipase A <sub>2</sub> Signaling Pathway in Blood—Brain Barrier Disruption after Focal Cerebral Ischemia and Reperfusion2008 · 125 citations
  5. 5Status Epilepticus: Risk Factors and Complications2000 · 267 citations