Case Report (n=1)
No
Atenolol provided temporary symptomatic relief in a miniature Bull Terrier with severe congenital mitral valve dysplasia and aortic stenosis, highlighting the challenges of managing complex congenital heart disease without surgical options.
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Documents biventricular hypertrophy with syncope in a young Bull Terrier; leaves open etiology, prevalence, and management questions in veterinary cardiology.
The clinical case of a one-year-old miniature Bull Terrier is presented. The patient was brought to the cardiology service of the Small Animal Clinic at the Universidad Nacional de Colombia due to historical syncopal episodes during exercise or moments of excitement, as well as at rest. Additionally, these syncopal episodes have increased in frequency to the point of daily occurrence. Echocardiography reveals thickening of the interventricular septum and the free wall of both ventricles. Additionally, irregular enlargement is evidenced in the mass of the papillary muscles of the left ventricle, which are distributed concentrically to the ventricular cavity, as well as a thickened mitral valve, which neither closes nor opens correctly causing mitral insufficiency and regurgitant flow to the left atrium. Similarly, narrowing of the left ventricular outflow tract and increased echogenicity in the aortic valve are observed, which, similarly to the mitral valve, is unable to open properly. The findings suggest congenital mitral valve dysplasia leading to stenosis, accompanied by aortic stenosis. Due to the absence of surgical intervention options, therapy aims to control existing clinical signs and halt the progression of cardiac enlargement using beta-blockers.
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Delgado et al. (2024) conducted a case report in Aortic stenosis and mitral valve dysplasia (n=1). Atenolol was evaluated on Frequency of syncopal episodes.
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