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October 26, 2001Circulation ResearchOpen Access

Antiischemic Effects of SB203580 Are Mediated Through the Inhibition of p38α Mitogen-Activated Protein Kinase

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Authors

JMJoel W. MartinMAMetin AvkiranRQRoy A. Quinlan

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Martin et al. (2001) studied this question.

synapsesocial.com/papers/6a915fd22bf604fc70c651fehttps://doi.org/10.1161/hh2101.099504
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Direct Inhibition of Cyclooxygenase-1 and -2 by the Kinase Inhibitors SB 203580 and PD 980591998 · 257 citations
  2. 2The Pyridinyl Imidazole Inhibitor SB203580 Blocks Phosphoinositide-dependent Protein Kinase Activity, Protein Kinase B Phosphorylation, and Retinoblastoma Hyperphosphorylation in Interleukin-2-stimulated T Cells Independently of p38 Mitogen-activated Protein Kinase2000 · 309 citations
  3. 3Cardiac Muscle Cell Hypertrophy and Apoptosis Induced by Distinct Members of the p38 Mitogen-activated Protein Kinase Family1998 · 854 citations
  4. 4Small Heat Shock Proteins and Protection Against Ischemic Injury in Cardiac Myocytes1997 · 345 citations
  5. 5Use of a drug‐resistant mutant of stress‐activated protein kinase 2a/p38 to validate the in vivo specificity of SB 2035801999 · 115 citations