Key result
A 58-year-old woman developed acquired long QT syndrome and torsades de pointes following an acute myocardial infarction, which was successfully treated with intravenous isoproterenol and magnesium.
Why the study?
This report serves to remind clinicians of acquired long QT syndrome and potentially fatal ventricular arrhythmia after myocardial infarction.
Case Report (n=1)
This case highlights the rare but potentially fatal occurrence of acquired long QT syndrome and torsades de pointes following acute myocardial infarction.
Answers are grounded in this paper and cite their sources.
MI may trigger acquired long QT syndrome without typical causes; single case leaves open mechanistic confirmation.
Acquired long QT syndrome is typically caused by medications, electrolyte disturbances, bradycardia, or catastrophic central nervous system events. We report a case of myocardial infarction-related acquired long QT syndrome in a 58-year-old woman that had no clear cause and progressed to torsades de pointes requiring treatment with isoproterenol and magnesium. Despite negative results of DNA testing against a known panel of genetic mutations and polymorphisms associated with long QT syndrome, the patient's family history of fatal cardiac disease suggests a predisposing genetic component. This report serves to remind clinicians of this potentially fatal ventricular arrhythmia after myocardial infarction.
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Gordon et al. (2020) conducted a case report in Acquired Long QT Syndrome after Acute Myocardial Infarction (n=1). Isoproterenol and magnesium was evaluated. A 58-year-old woman developed acquired long QT syndrome and torsades de pointes following an acute myocardial infarction, which was successfully treated with intravenous isoproterenol and magnesium.
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