nasal cultures in 6 of the 11 members of the 2 families.In this niche, it was able to persist and cause a series of infections in a relatively large number of family members.Even though the S. aureus isolated from active lesions were not available for testing, the recovery of identical PVL-positive organisms from nasal cultures strongly suggests the presence of a pathogenic clone that probably caused the recurrent infections in the 6 affected family members.Our investigation highlights the high transmissibility of this PVL-producing S. aureus clone, its high attack rate, and its virulence.The intervention in this outbreak might have prevented not only subsequent recurrences of cutaneous infections but also further spread of this clone and the manifestation of even more serious infections such as necrotizing pneumonia.Increasing awareness among community-based healthcare providers of PVL-producing S. aureus infections is important to facil-
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Lee et al. (2006) studied this question.
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