Key result
Swimming stress produced a marked increase in adrenaline content in the heart, spleen, and submaxillary gland of rats, which was abolished by pretreatment with desmethylimipramine.
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Does not support clinical use in stress modulation; leaves open noradrenergic mechanisms in rodent stress models pending higher-evidence studies.
Ayako SUDO (1985) studied this question. Swimming stress vs. Control (basal conditions) was evaluated on Adrenaline and noradrenaline content in the heart, spleen, and submaxillary gland. Swimming stress produced a marked increase in adrenaline content in the heart, spleen, and submaxillary gland of rats, which was abolished by pretreatment with desmethylimipramine.
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