Key result
Seneca Valley Virus 3C protease mediates the cleavage and redistribution of nucleolin to facilitate viral replication.
Authors
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NCL may be an antiviral target; leaves open any role in virus-related cardiovascular disease.
Key points are not available for this paper at this time.
Song et al. (2022) studied this question. Seneca Valley Virus 3C protease mediates the cleavage and redistribution of nucleolin to facilitate viral replication.
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