The detection of a bovine P[14] allele closely related to human P[14] strains suggests that human G6P[14] and G10P[14] rotaviruses originated from reassortment events involving bovine strains.
Supports zoonotic surveillance in rotavirus monitoring; leaves open frequency and clinical relevance of bovine-human reassortment.
Earlier studies have proposed that human G6P\[14\] (PA169-like VP7), G8P[14], and G10P[14] group A rotavirus strains resulted from interspecies transmission between humans and cattle ([1][1], [4][2], [9][3], [11][4], [13][5], [14][6]). However, to our knowledge, PA169-like G6 strains have not been
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Ghosh et al. (2007) studied this question.