Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
April 8, 2019Cancer Cell InternationalOpen Access

Systems biology based meth-miRNA–mRNA regulatory network identifies metabolic imbalance and hyperactive cell cycle signaling involved in hepatocellular carcinoma onset and progression

View Full Paper
Ask AI
Bookmark
Share

Authors

KZKashif Rafiq ZahidUniversity of Alabama at BirminghamMSMing‐Yang SuQinghai UniversityAKAbdur Rehman Raza KhanNational University of Sciences and Technology

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Zahid et al. (2019) studied this question.

synapsesocial.com/papers/6a952f4b035d31caad2c906ahttps://doi.org/10.1186/s12935-019-0804-3
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The microRNAs miR-200b-3p and miR-429-5p target the LIMK1/CFL1 pathway to inhibit growth and motility of breast cancer cells2017 · 91 citations
  2. 2p16INK4 is inactivated by extensive CpG methylation in human hepatocellular carcinoma1999 · 204 citations
  3. 3Clinical Significance and Prognostic Value of microRNA Expression Signatures in Hepatocellular Carcinoma2013 · 104 citations
  4. 4High expression of miR-105-1 positively correlates with clinical prognosis of hepatocellular carcinoma by targeting oncogene NCOA12017 · 43 citations
  5. 5Exploring genome-wide DNA methylation profiles altered in hepatocellular carcinoma using Infinium HumanMethylation 450 BeadChips2012 · 154 citations