Key result
Pretreatment with 1,6-bis(phosphocholine)-hexane inhibits increased myocardial damage resulting from human C-reactive protein in a rat model of coronary-artery occlusion.
Why the study?
Does pretreatment with 1,6-bis(phosphocholine)-hexane reduce myocardial damage in a rat model of coronary-artery occlusion?
Does pretreatment with 1,6-bis(phosphocholine)-hexane reduce myocardial damage in a rat model of coronary-artery occlusion?
Inhibition of C-reactive protein with 1,6-bis(phosphocholine)-hexane reduces myocardial damage in a rat model of acute myocardial infarction.
Answers are grounded in this paper and cite their sources.
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May attenuate CRP-mediated injury in experimental MI; leaves open translation to human acute coronary syndromes.
A recent study in a rat model of coronary-artery occlusion shows that pretreatment with 1,6-bis(phosphocholine)-hexane inhibits increased myocardial damage that results from human C-reactive protein.
Kitsis et al. (2006) studied this question. Pretreatment with 1,6-bis(phosphocholine)-hexane inhibits increased myocardial damage resulting from human C-reactive protein in a rat model of coronary-artery occlusion.