Key result
Double knockout of MRTF-A and MRTF-B in mice resulted in abnormally small and disorganized sarcomeres, causing skeletal muscle hypoplasia and perinatal lethality.
MRTFs are essential for sarcomere formation and actin cycling during skeletal muscle development, and their absence leads to skeletal muscle hypoplasia and perinatal lethality.
Mouse MRTF-A/B double knockout implicates these factors in sarcomere assembly; leaves open any role in human myopathies or therapeutic targeting.
Myocardin-related transcription factors (MRTFs) play a central role in the regulation of actin expression and cytoskeletal dynamics. Stimuli that promote actin polymerization allow for shuttling of MRTFs to the nucleus where they activate serum response factor (SRF), a regulator of actin and other cytoskeletal protein genes. SRF is an essential regulator of skeletal muscle differentiation and numerous components of the muscle sarcomere, but the potential involvement of MRTFs in skeletal muscle development has not been examined. We explored the role of MRTFs in muscle development in vivo by generating mutant mice harboring a skeletal muscle-specific deletion of MRTF-B and a global deletion of MRTF-A. These double knockout (dKO) mice were able to form sarcomeres during embryogenesis. However, the sarcomeres were abnormally small and disorganized, causing skeletal muscle hypoplasia and perinatal lethality. Transcriptome analysis demonstrated dramatic dysregulation of actin genes in MRTF dKO mice, highlighting the importance of MRTFs in actin cycling and myofibrillogenesis. MRTFs were also shown to be necessary for the survival of skeletal myoblasts and for the efficient formation of intact myotubes. Our findings reveal a central role for MRTFs in sarcomere formation during skeletal muscle development and point to the potential involvement of these transcriptional co-activators in skeletal myopathies.
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Cenik et al. (2016) studied Skeletal muscle development. Skeletal muscle-specific deletion of MRTF-B and global deletion of MRTF-A was evaluated on Sarcomere formation and muscle development. Double knockout of MRTF-A and MRTF-B in mice resulted in abnormally small and disorganized sarcomeres, causing skeletal muscle hypoplasia and perinatal lethality.
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