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September 1, 2026BiomedicinesOpen Access

Frequencies of CYP2C19 Polymorphisms in Mexican Mestizo Patients with Coronary or Cerebral Atherosclerosis and Their Association with Residual Platelet Activity on Clopidogrel Treatment

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Why the study?

Clopidogrel response shows wide individual variability linked to CYP2C19 polymorphisms, and clopidogrel resistance relates to adverse outcomes in CAD, but prevalence and effects in Mexican mestizo patients needed evaluation.

Do CYP2C19 polymorphisms predict clopidogrel resistance in Mexican mestizo patients with coronary or cerebral atherosclerosis?

Population

160 CAD and 11 stroke Mexican mestizo patients treated with clopidogrel

Comparison

Unfavorable vs favorable CYP2C19 metabolizer genotypes

Design

Observational study

Key result

Unfavorable CYP2C19 genotypes showed a non-significant trend toward higher clopidogrel resistance compared to favorable genotypes (64.7% vs 39.1%; p=0.059).

Authors

RIRaúl Izaguirre-ÁvilaBVBeatriz Villegas-TorresECEvelyn Cortina-de-la-Rosa

Discussion

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Member takes

Overview

CYP2C19 variants may predict clopidogrel response in Mexican mestizos; leaves open whether genotyping improves outcomes in this population.

Study Design

Type

Observational (n=171)

Structured PICO

Do CYP2C19 polymorphisms predict clopidogrel resistance in Mexican mestizo patients with coronary or cerebral atherosclerosis?

P
Population
171 Mexican mestizo patients with coronary artery disease or stroke receiving clopidogrel, of whom 81 were tested for platelet aggregation.
E
Exposure
CYP2C19 genotyping (*1, *2, *3, and *17 alleles) and platelet aggregation testing
C
Comparator
Favorable genotypes (CYP2C19 *1/*1, *1/*17, and *17/*17) vs. unfavorable genotypes for metabolism (CYP2C19 *1/*2, *2/*17, and *2/*2)
O
Outcome
Prevalence of CYP2C19 polymorphisms and clopidogrel resistance assessed by ADP-induced aggregation in platelet-rich plasmasurrogate

Main Result

Absolute Event Rate: 64.7% vs 39.1%

p-value: p=0.059

In Mexican mestizo patients, CYP2C19 polymorphisms alone do not fully explain in vitro clopidogrel resistance, which appears to be a complex multifactorial phenomenon.

Cite This Study

Izaguirre-Ávila et al. (2026) conducted an observational in Coronary artery disease or stroke (n=171). Unfavorable CYP2C19 genotypes (*1/*2, *2/*17, and *2/*2) vs. Favorable CYP2C19 genotypes (*1/*1, *1/*17, and *17/*17) was evaluated on Clopidogrel resistance (p=0.059). Unfavorable CYP2C19 genotypes showed a non-significant trend toward higher clopidogrel resistance compared to favorable genotypes (64.7% vs 39.1%; p=0.059).

synapsesocial.com/papers/6a9926b3f3ae593e0136346fhttps://doi.org/10.3390/biomedicines14091970
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Cytochrome P-450 CYP2C19 genetic polymorphism and its relation with clopidogrel resistance2023 · 3 citations
  2. 2Clopidogrel resistance and its relevance: Current concepts2024 · 12 citations
  3. 3Effects of CYP2C19 Genotype on Outcomes of Clopidogrel Treatment2010 · 538 citations
  4. 4An improved method for specific and quantitative determination of the clopidogrel active metabolite isomers in human plasma2011 · 96 citations
  5. 5Multicenter evaluation of the safety and efficacy of varying doses of cangrelor used in acute cerebrovascular stenting in patients with acute ischemic stroke2025 · 1 citations