Key result
FPR2 and THBS3 genes may drive the pathogenesis of coronary slow flow disease through immune-related pathways such as antigen processing and Th17 cell differentiation.
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Genes such as FPR2 and THBS3 may play important roles in the pathogenesis of SCF through immune-related pathways such as antigen processing and presentation and Th17 cell differentiation.
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Sun et al. (2021) studied this question. FPR2 and THBS3 genes may drive the pathogenesis of coronary slow flow disease through immune-related pathways such as antigen processing and Th17 cell differentiation.
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