KRATZER et al. (1959) observed that turkey poults, fed an isolated soybean protein diet, recovered from the zinc deficiency when small amounts of EDTA (ethylenediaminetetraacetic acid) were fed in the ration. Similar effects of EDTA have been reported for chickens (O’Dell et al., 1964) and for rats (Oberleas et al., 1966). EDTA was reported to decrease iron availability to rats (Larsen et al., 1960) and to reduce cobalt toxicity in chickens (Turk and Kratzer, 1960). The nature of this chelation was studied by Vohra and Kratzer (1964) and by Nielsen et al. (1966) using different chelating agents. They found that the availability of zinc was increased as the stability constants of the chelating agents increased. The greatest response to the chelating agents was obtained when sequestrenes having stability constants between 14 and 15 were fed. Growth rate and the healing of zinc deficiency symptoms decreased as the stability constant values . . .
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Suso et al. (1968) studied this question.