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Fracture repair is a complex, specialized process whereby bone regenerates to restore pre-injury function to the skeleton. Despite successful healing in the majority of cases, a significant number of fractures require further treatment for delayed union or nonunion. As our knowledge of the molecular mechanisms that govern fracture repair continues to improve, new strategies for developing biologically based therapies will follow. This review examines the molecular aspects of adult skeletogenesis.
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Miclau et al. (2000) studied this question.
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