Key result
Neonatal Brachial Plexus Paralysis, primarily caused by dystocic delivery, has a favorable prognosis with 80% of C5-C6 lesions recovering spontaneously, though 20-30% of patients develop persistent deficits.
Neonatal brachial plexus paralysis is primarily managed conservatively with physical therapy, with surgical intervention indicated for patients lacking biceps recovery by 3 to 6 months.
Supports conservative PT-first management in neonatal C5-C6 palsy; leaves open optimal surgical thresholds without prospective data.
Neonatal Brachial Plexus Paralysis (PPBN) is defined as a brachial plexus traction or compression injury resulting from dystocic delivery. The incidence of PPBN varies from 0.42 to 5.1% per 1000 births. The lesion occurs in the expulsive period of childbirth, is most often associated with shoulder dystocia, gestational or pre-gestational diabetes and macrosomic fetuses. The lesion resulting from C5-C6 root involvement is the most frequent, affecting around 50 to 60% of the cases, and 80% of these patients have spontaneous recovery. However, around 20 to 30% of patients may develop persistent deficits, with permanent impacts on upper limb function, compromising the child's development; leading to repercussions for their families and for the health system. PPBN is usually diagnosed by clinical examination and usually does not require imaging studies. Initial treatment is conservative based on functional rehabilitation through physical therapy. Sequential physical examination will indicate patients with no recovery of shoulder movement between 3 and 6 months, who may benefit from surgical treatment. The prognosis of PPBN depends on the level (pre- or postganglionic), the extent, severity of the lesion, the speed of recovery and the quality of initial clinical management.
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Brito et al. (2019) conducted a review in Neonatal Brachial Plexus Paralysis. Management of Neonatal Brachial Plexus Paralysis was evaluated. Neonatal Brachial Plexus Paralysis, primarily caused by dystocic delivery, has a favorable prognosis with 80% of C5-C6 lesions recovering spontaneously, though 20-30% of patients develop persistent deficits.
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