Splanchnic ethanol uptake was lower in cirrhosis compared to normals. The accelerating effect of fructose on ethanol metabolism was smaller in cirrhosis. From studies of portocaval shunts, it appeared that acetate, formed from ethanol, was not metabolized in the liver. The peripheral metabolism of acetate, glucose and lactate was diminished in cirrhosis.
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Winkler et al. (1969) studied this question.
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