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March 4, 2003Journal of Medical Virology

Absence of SLAM mutations in EBV‐associated lymphoproliferative disease patients

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Authors

VFVéronique FerrandCentre international de recherche sur le cancerCLCuiling LiJinzhou Medical UniversityGRGiovanni RomeoIRCCS Azienda Ospedliero-Universitaria di Bologna Policlinico di Sant'Orsola

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Ferrand et al. (2003) studied this question.

synapsesocial.com/papers/6a9e31fcaa8062d6ddbe5fa6https://doi.org/10.1002/jmv.10373
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1SLAM and its role in T cell activation and Th cell responses1997 · 100 citations
  2. 2Gntb-A, a Novel Sh2d1a-Associated Surface Molecule Contributing to the Inability of Natural Killer Cells to Kill Epstein-Barr Virus–Infected B Cells in X-Linked Lymphoproliferative Disease2001 · 306 citations
  3. 3Epstein--Barr Virus-Negative Boys With Non-Hodgkin Lymphoma Are Mutated in the Sh2D1A Gene, as Are Patients With X-Linked Lymphoproliferative Disease (XLP)1999 · 108 citations
  4. 4Molecular and Functional Characterization of Mouse Signaling Lymphocytic Activation Molecule (SLAM): Differential Expression and Responsiveness in Th1 and Th2 Cells1999 · 110 citations
  5. 5Diagnosis of X-linked lymphoproliferative disease by analysis of SLAM-associated protein expression2000 · 66 citations