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The recruitment of neutrophils to sites of inflammation, their battle against invading microorganisms through phagocytosis and the release of antimicrobial agents is a highly coordinated and tightly regulated process that involves the interplay of many different receptors, ion channels and signalling pathways. Changes in intracellular calcium levels, caused by cytosolic Ca 2+ store depletion and the influx of extracellular Ca 2+ via ion channels, play a critical role in synchronizing neutrophil activation and function. In this review, we provide an overview of how Ca 2+ signalling is initiated in neutrophils and how changes in intracellular Ca 2+ levels modulate neutrophil function.
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Immler et al. (2018) studied this question.
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