Key result
Postprandial cardiac output increased by 61% at rest and 23% during exercise, changes that were largely dependent on the autonomic nervous system.
Why the study?
Does autonomic blockade of the heart alter postprandial hemodynamic changes at rest and during upright exercise in healthy subjects?
Does autonomic blockade of the heart alter postprandial hemodynamic changes at rest and during upright exercise in healthy subjects?
Most postprandial changes in the central circulation, including increases in cardiac output at rest and during exercise, are under the control of the autonomic nervous system.
Answers are grounded in this paper and cite their sources.
Supports autonomic dependence of postprandial hemodynamics; hypothesis-generating and should not yet alter practice.
Postprandial hemodynamic changes were studied in healthy subjects at rest and during exercise in the upright position with and without autonomic blockade of the heart. At rest cardiac output increased 61% mostly because of a stroke volume increase accomplished by left ventricular end-diastolic dilation. These changes seemed to be dependent on the autonomic nervous system, whereas the postprandial heart rate increase did not. During exercise cardiac output was 23% higher after food intake due to a rise in both stroke volume and heart rate. These changes were apparently under influence of the autonomic nervous system, whereas left ventricular dilation was not. The present findings indicate that most of the postprandial changes in the central circulation are under control of the autonomic nervous system.
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Kelbæk et al. (1987) studied Healthy subjects. Autonomic blockade of the heart vs. Without autonomic blockade was evaluated on Postprandial hemodynamic changes (cardiac output, stroke volume, heart rate). Postprandial cardiac output increased by 61% at rest and 23% during exercise, changes that were largely dependent on the autonomic nervous system.
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