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September 1, 2008Molecular Cancer TherapeuticsOpen Access

17α-Hydroxylase/17,20 lyase inhibitor VN/124-1 inhibits growth of androgen-independent prostate cancer cells via induction of the endoplasmic reticulum stress response

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Authors

RBRobert D. BrunoDominion University CollegeTGTony D. GoverUniversity of Maryland, BaltimoreABAngelika M. BurgerCancer Clinic

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Bruno et al. (2008) studied this question.

synapsesocial.com/papers/6a9fe383c2a6bb3d9844af9chttps://doi.org/10.1158/1535-7163.mct-08-0336
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Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Novel Steroidal Inhibitors of Human Cytochrome P45017.alpha.-Hydroxylase-C17,20-lyase): Potential Agents for the Treatment of Prostatic Cancer1995 · 381 citations
  2. 2Quantification of P450scc, P45017α, and Iron Sulfur Protein Reductase in Leydig Cells and Adrenals of Inbred Strains of Mice*1988 · 65 citations
  3. 3Integrated Endoplasmic Reticulum Stress Responses in Cancer2007 · 439 citations