Why the study?
Spontaneous coronary artery dissection is relatively rare and frequently misdiagnosed, with limited knowledge regarding its pathophysiology and management.
Case Report (n=1)
No
Discontinuation of anticoagulation in a patient with recurrent SCAD led to clinical worsening, highlighting the uncertainty in optimal antithrombotic management for this condition.
Worsening of recurrent SCAD after anticoagulation stop; leaves open prolonged anticoagulation role and questions antiplatelet-centric management.
Spontaneous coronary artery dissection (SCAD) is a relatively rare and frequently misdiagnosed disease. The current knowledge of its pathophysiology and management is limited and based mostly on hypotheses. We present a patient with recurrent SCAD whose condition worsened soon after discontinuation of anticoagulation, prompting us to question the current management and review the evidence about pathophysiology, anticoagulation, and antiplatelet therapy.
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Sanchez et al. (2022) conducted a case report in Spontaneous coronary artery dissection (SCAD) (n=1). Anticoagulation (heparin drip) vs. Discontinuation of anticoagulation was evaluated on Clinical improvement (resolution of chest pain and down-trending troponins).
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