Little has been added to the knowledge of the treatment of renal rickets since the paper by Graham and Oakley (1938), and the reader is referred to their work for a summary of the previous literature of this condition. Whether or not in fact rickets is the principal bony lesion has been questioned by Langmead and Orr (1933). From a morbid anatomical study, they concluded that osteitis fibrosa was the outstanding pathological change in the bones, and attributed this to *osteoclastic activity caused by excessive parathyroid function. They were able to demonstrate the presence of hyper-trophy of the parathyroid glands. Their view has received support in America from such authorities as Park and Eliot (1942), and Hamilton (1940) who
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W. Sheldon (1943) studied this question.
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