Experimental study demonstrates enhanced iron uptake by intestinal cells via reduced iron oxidation, indicating optimized therapeutic strategies for iron deficiency anemia.
< 0.05). Our data points to formulated curcumin's ability to limit oxidation of divalent iron as a possible mechanism to explain the greater iron uptake by intestinal cells. This study characterises for the first time the distinct interaction behaviour of curcumin forms with iron and the potential consequences on iron uptake and intestinal health, providing novel insights that may help optimise therapeutic approaches to tackle ID and IDA.
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Gámez-Fernández et al. (2025) studied this question.
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