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Abstract β-Bungarotoxin (β-BuTX) induces twitching of innervated frog muscle and subsequently blocks transmitter release from motor nerve endings. These actions of β-BuTX are prevented if the nerve-muscle junctions are pretreated with a low concentration of phospholipase A2 from bee venom. When the Ca2+ in the external fluid is replaced by Sr2+, the phospholipase activity of β-BuTX is negligible and, in these conditions, β-BuTX causes a decrease in the amplitude of endplate potentials but does not go on to block transmitter release completely.
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Abe et al. (1978) studied this question.
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