Key result
Pediatric case reveals transient vascular impairment during self-induced stretch syncope, supporting a multifactorial etiology.
Why the study?
Stretch syncope is an uncommon condition mainly described in adolescent males, with few reported cases and uncertain pathogenesis involving local and systemic hemodynamic phenomena.
Case Report (n=1)
This case report provides new insights into the neurophysiological profile of stretch syncope, suggesting it is a multifactorial disorder involving transient vascular impairment.
Answers are grounded in this paper and cite their sources.
May warrant vascular assessment in pediatric stretch syncope; leaves open the multifactorial hypothesis pending larger studies.
Stretch syncope (SS) is a benign, uncommon, distinct condition described mainly in adolescent males. It is responsible for paroxysmal events started by stereotyped stretching actions with neck hyperextension, culminating in alteration of consciousness. Motor manifestations are often present and may be associated with a generalized slowing of the electroencephalographic activity, challenging the diagnosis. Despite a few cases reported in the literature, different mechanisms have been implied in the pathogenesis, involving both local and systemic hemodynamic phenomena. Here, we report on an 8-year-old girl with self-induced SS, providing new insights into the related neurophysiological profile and discussing the possible etiology. Our evidence of transient and dynamic vascular impairment supports the hypothesis of SS as a multifactorial disorder.
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Mercante et al. (2024) conducted a case report in Self-induced stretch syncope (n=1). Self-induced stretch syncope was evaluated. An 8-year-old girl with self-induced stretch syncope demonstrated transient and dynamic vascular impairment, supporting the hypothesis of stretch syncope as a multifactorial disorder.
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