Key result
Diet-supplemented EPA leads to the regression of atherosclerotic lesions in the murine Ldlr -/- model by inducing indoleamine 2,3-dioxygenase and downregulating costimulatory molecules.
Why the study?
Does diet-supplemented eicosapentaenoic acid (EPA) cause regression of atherosclerotic lesions in a murine Ldlr -/- model?
Population
murine Ldlr -/- model
Design
Editorial
Authors
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EPA supplementation may promote regression of atherosclerotic lesions through immune modulation involving dendritic cells and T lymphocytes.
Does diet-supplemented eicosapentaenoic acid (EPA) cause regression of atherosclerotic lesions in a murine Ldlr -/- model?
EPA supplementation may promote regression of atherosclerotic lesions through immune modulation involving dendritic cells and T lymphocytes.
Lau et al. (2011) conducted an editorial in Atherosclerotic lesions. Diet-supplemented Eicosapentaenoic acid (EPA) was evaluated on Regression of atherosclerotic lesions. Diet-supplemented EPA leads to the regression of atherosclerotic lesions in the murine Ldlr -/- model by inducing indoleamine 2,3-dioxygenase and downregulating costimulatory molecules.
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