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August 12, 2026SLAS TECHNOLOGYOpen Access

MINPP1, HES4, and SLCO5A1 emerge as core genes linking T2D to adverse CABG outcomes.

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Why the study?

To identify signature genes mediating the effects of type 2 diabetes on CABG and elucidate their molecular mechanisms and therapeutic value.

Population

Multi-cohort T2D and CABG datasets, clinical CABG patients, and human aortic smooth muscle cells

Design

Bioinformatics analysis, machine learning, clinical validation, and in vitro experimental study

Key result

MINPP1, HES4, and SLCO5A1 were identified as core signature genes mediating the adverse effects of type 2 diabetes on coronary artery bypass grafting, with diagnostic AUC values > 0.625.

Authors

GWGang WangZFZunyi FengSKShuba Kang

Discussion

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Overview

Does not yet support gene-based risk tools in diabetic CABG; leaves open prospective validation of these signatures.

Structured PICO

P
Population
Multi-cohort type 2 diabetes (T2D) and coronary artery bypass grafting (CABG) datasets, clinical CABG patients, and human aortic smooth muscle cells
E
Exposure
Bioinformatics screening, clinical validation, and in vitro SLCO5A1 knockdown
C
Comparator
Control groups (non-T2D or control cells)
O
Outcome
Identification of core signature genes mediating the effects of T2D on CABG and their molecular regulatory mechanismssurrogate

MINPP1, HES4, and SLCO5A1 are identified as key signature genes mediating the adverse effects of type 2 diabetes on CABG outcomes, offering potential novel therapeutic targets.

Cite This Study

Wang et al. (2026) studied Type 2 diabetes and coronary artery bypass grafting. Type 2 diabetes was evaluated on Identification of core signature genes mediating the adverse effects of T2D on CABG. MINPP1, HES4, and SLCO5A1 were identified as core signature genes mediating the adverse effects of type 2 diabetes on coronary artery bypass grafting, with diagnostic AUC values > 0.625.

synapsesocial.com/papers/6aa4704cac77d5296f2bc31chttps://doi.org/10.1016/j.slast.2026.100459
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