Key result
Hyperparathyroidism in hypophosphatemic rickets is linked to severe cardiac calcifications and arrhythmias despite normal calcium.
Case Report (n=1)
Highlights the risk of ectopic cardiac calcification and valve dysfunction due to hyperparathyroidism in patients treated for hypophosphatemic rickets, emphasizing the need for parathyroid monitoring.
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May warrant parathyroid monitoring in hypophosphatemic rickets; single case leaves open causal role and screening needs.
Moltz et al. (2001) conducted a case report in Hypophosphatemic rickets and hyperparathyroidism (n=1). Hyperparathyroidism was evaluated on Cardiac calcifications leading to aortic and mitral valve dysfunction, myocardial calcification, and arrhythmia. Hyperparathyroidism in an adolescent with hypophosphatemic rickets was associated with severe cardiac calcifications, valve dysfunction, and arrhythmia despite normal calcium levels.
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